By Elaine A. Moore
In 2001 Graves' disorder: a realistic advisor defined the reasons, analysis, remedy and affliction process Graves' disorder and different hyperthyroid issues, reminiscent of poisonous multinodular goiter, thyroiditis, resistance to thyroid hormone, and hyperthyroidism brought on by drugs and genetic mutations. the current paintings maintains the above yet specializes in next advances in affliction pathology, together with discoveries in regards to the genetic, immune approach, and environmental elements that bring about hyperthyroid issues; new instructions for traditional remedy; and replacement and complementary scientific treatments. extra sections describe targeted situations reminiscent of hyperthyroidism in being pregnant and in young ones and temporary hyperthyroidism within the infant.
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Extra info for Advances in Graves' Disease and Other Hyperthyroid Disorders
The characteristic ﬁndings include an elevated TSH level, elevated thyroid hormone levels, and an elevated alpha subunit of TSH, measured by specialized laboratory procedures. Although TSHomas have been found in children, they are more common in elderly patients. Excess TRH Production Another disorder that causes excess TSH production and thyrotoxicosis is the rare condition of excess thyrotropin releasing hormone (TRH), which is also known as pituitary T3 resistance. TRH hypersecretion is characterized by an absence of pituitary tumor, elevated TSH levels and failure to respond to TRH administration.
In the conditions of hyperthyroidism described in the following sections, this feedback mechanism is overcome by other factors that cause the thyroid gland to produce excess thyroid hormone regardless of a drastic drop in the TSH level. Graves’ Disease Graves’ disease stands out as the most common cause of hyperthyroidism, accounting for approximately 80 percent of all cases (Nussey and Whitehead 2001, 6). The autoimmune mechanism is described brieﬂy here and the disease process in Graves’ disease is addressed in Chapter 3.
This results in hypothyroidism. SUBACUTE THYROIDITIS (SAT) Subacute painful thyroiditis (SAT), which is also known as DeQuervain thyroiditis and granulomatous painful thyroiditis, was ﬁrst described by DeQuervain in 1940. SAT causes granulomatous-type changes and giant cells in thyroid glands of those affected. SAT is fairly common and may account for as many as 5 percent of all visits to physicians for thyroid abnormalities (Singer 1991, 63). Thought to have a viral origin, SAT frequently follows a respiratory infection and is most often seen in the summer and fall.