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Download e-book for kindle: Aluminum and renal failure by Marc E. de Broe, Patrick C. D’Haese (auth.), Marc E. de

By Marc E. de Broe, Patrick C. D’Haese (auth.), Marc E. de Broe, Jack W. Coburn (eds.)

` ... the cloth awarded presents priceless info in regards to the organic results and scientific syndromes linked to aluminum publicity. This quantity could be precious for college kids and nephrologists who're drawn to furthering their realizing of the area.'
Dialysis and Transplantation, 20/1, January 1991

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Extra info for Aluminum and renal failure

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Karlik SJ, Tarien E, Elgavish GA, Eichhorn GL, 1983: Aluminum-27 nuclear magnetic resonance study aluminum(III) interactions with carboxylate ligands. Inorg Chern 22: 525. 28. Martin RB, Savory J, Brown S, Bertholf RL, and Wills MR, 1987: Transferrin binding of AI3+ and Fe 3 +. Clin Chern 33: 405. 29. Macdonald TL, Humphreys WG, and Martin RB, 1987: Promotion of tubulin assembly by aluminum ion in vitro. Science 236: 183. 30. Martin RB, 1986: Citrate binding of Ae+ and Fe 3 +. J Inorg Biochem 28: 181.

A dialysis patient with severe aluminum overload without apparent clinical toxicity [48], we consider liver sequestration of aluminum to be an important mechanism in neutralizing aluminum's deleterious effects. Effect of age With exception of the lung [3] and the brain [33] very few data are found in the literature reporting increases of aluminum levels with age. However, many reports deal with increased toxicity from orally administered aluminum in children. This could either be due to relatively higher doses necessary to maintain the phosphate levels in these subjects, to a better compliance or to an increased aluminum absorption in the immature gut [43].

The initial studies were concerned with pediatric patients with renal disease on oral Al medication which developed neurological symptoms, in the absence of dialysis treatment. These symptoms were tentatively diagnosed as - analogous to - dialysis encephalopathy while their correlation to oral Al medication could only be implied [43-47]. The growing awareness of the possible involvement of Al in these neurological symptoms lead other investigators to analyze tissue and plasma levels of Al to support the diagnosis of Al toxicity and to prove intestinal absorption of AI.

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